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PDF Availablehttps://doi.org/10.1186/gb-2004-5-7-r47
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Cite Journal: Genome Biology |  Publication Date: Jan 1, 2004 |
 Citations: 131 |  License type: NO-CC CODE |
Human disease genes differ significantly from their rodent orthologs with respect to their overall levels of conservation and their rates of evolutionary change. Rodent orthologs of human trinucleotide repeat-expansion disease genes were also found to contain substantially fewer such repeats.
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