Abstract
Fixation of CO 2 to the β-methyl group has been postulated as a pathway for the catabolism of β-methyl-substituted fatty acids in mammalian tissue. In the present paper the degradation of all-over tritium-labeled 3,6-dimethyl-octanoic acid by rat kidney slices in the presence of 14CO 2 is described. No double labeling of acetate or other intermediates could be detected. It is concluded that no CO 2-fixation has taken place in the degradation of this β-methyl-substituted fatty acid.
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