Abstract
Aim: Inhibition of the mechanistic target of rapamycin (mTOR) is a promising approach to halt atherogenesis in different animal models. This study aims to elucidate mechanisms involved in the anti-atherosclerotic effects of the mTOR inhibitor everolimus using ApoE-/- mice with a heterozygous mutation in the fibrillin-1 gene (Fbn1C1039G+/-), representing a novel model of advanced atherosclerosis.
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