Abstract
This work was to study the application value of dynamic electrocardiogram (ECG) feature data in evaluating the curative effect of percutaneous coronary intervention in acute ST-segment elevation myocardial infarction with hypertension, so as to facilitate the early diagnosis and treatment of the disease. In this study, 90 patients with acute ST-segment elevation myocardial infarction accompanied by hypertension were selected as the study subjects and randomly divided into group A (oral aspirin antiplatelet therapy), group B (thrombolytic drug streptokinase (SK) therapy), and group C (percutaneous coronary intervention), with 30 cases in each group. In addition, a P-wave detection algorithm was introduced for automatic detection and analysis of electrocardiograms, and the efficacy of patients was assessed by Holter feature data based on the P-wave detection algorithm. The results showed that the diagnostic error rate, sensitivity, and predictive accuracy of the P-wave detection algorithm for ST-segment elevation myocardial infarction caused by acute occlusion of left main coronary artery (LMCA) were 0.24%, 95.41%, and 92.33%, respectively; the diagnostic error rate, sensitivity, and predictive accuracy for non-LMCA (nLMCA) ST-segment elevation myocardial infarction were 0.28%, 95.32%, and 96.07%, respectively; the proportion of patients with symptom to blood flow patency time <3 h in group C (55.3%) was significantly higher than that in groups A and B (22.1% and 22.6%) ( P < 0.05). Compared with group A, the content of B-type natriuretic peptide (pre-proBNP) at 1 week, 2 weeks, and 3 weeks after treatment in groups B and C was significantly lower and group C was significantly lower than group B ( P < 0.05). In summary, the P-wave detection algorithm has a high application value in the diagnosis and early prediction of acute ST-segment elevation myocardial infarction. Percutaneous coronary intervention in the treatment of acute ST-segment elevation myocardial infarction with hypertension can shorten the opening time of infarction blood flow, so as to effectively protect the heart function of patients.
Highlights
Acute ST-segment elevation myocardial infarction refers to acute myocardial ischemic necrosis, most of which occur on the basis of coronary artery disease with a sharp decrease in coronary blood supply or complete interruption, resulting in severe and lasting acute myocardial ischemia [1]. e common reason is secondary thrombosis based on unstable plaque rupture and erosion of coronary artery, resulting in continuous and complete occlusion of coronary artery. e disease was common in Europe and the United States. e annual incidence of the disease in the population aged 35–84 years old in the United States was about 7.1%
ECG Manifestations of ST-Segment Elevation Myocardial Infarction. ere was no typical single curve elevation of ST-segment and Q-wave formation in the early stage of ST-segment elevation myocardial infarction. ere were only hyperacute injury changes such as increased T-wave broadening and mirror changes in corresponding leads. e early ECG of ST-segment elevation myocardial infarction showed elevated T-wave, widened base, increased amplitude, and accompanied by asymmetric ascending and descending branches of T-wave lasting for several minutes
For patients with chest pain suspected of acute ST-segment elevation myocardial infarction, 12-lead electrocardiogram should be recorded within 10 minutes after the first medical exposure, and V3R–V5R and V7–V9 leads should be added to myocardial infarction [17, 18]. e early ECG of typical patients with acute STsegment elevation myocardial infarction showed that the ST-segment arch was elevated with or without pathological Q-wave and R-wave reduction
Summary
Acute ST-segment elevation myocardial infarction refers to acute myocardial ischemic necrosis, most of which occur on the basis of coronary artery disease with a sharp decrease in coronary blood supply or complete interruption, resulting in severe and lasting acute myocardial ischemia [1]. e common reason is secondary thrombosis based on unstable plaque rupture and erosion of coronary artery, resulting in continuous and complete occlusion of coronary artery. e disease was common in Europe and the United States. e annual incidence of the disease in the population aged 35–84 years old in the United States was about 7.1%. Acute ST-segment elevation myocardial infarction refers to acute myocardial ischemic necrosis, most of which occur on the basis of coronary artery disease with a sharp decrease in coronary blood supply or complete interruption, resulting in severe and lasting acute myocardial ischemia [1]. For the treatment of acute ST-segment elevation myocardial infarction, drug thrombolysis therapy (plasminogen activator to activate fibrinogen in thrombus, so as to transform into fibrinolysin and dissolve thrombus in coronary artery) and percutaneous coronary intervention. Percutaneous coronary intervention after thrombolysis can achieve reperfusion to restore blood flow as early as possible and strive for more reperfusion time for transport percutaneous coronary intervention, which has been widely recognized by domestic and foreign guidelines and is a feasible and suitable treatment strategy for ST-segment elevation myocardial infarction patients in China [9, 10]
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