Abstract
Serotonin and cAMP can facilitate synaptic transmission between sensory and motor neurons mediating the gill withdrawal reflex. The increase in cAMP would enhance the mobilization of transmitter release by increasing the free Ca++ in the terminals acting on the Ca conductance of the external membrane of the terminals or on Ca++ pool of subcellular organelles. The recent studies extended these findings to the nervous system of leech, Hirudo medicinalis. It was found that electrophysiological and neurochemical experiments takes into account for an involvement of serotonin and cAMP formation correlated with short-term plastic changes in a fast-conducting system in the ganglia of the leeches.
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
