Abstract
The periodontal disease (PD) is triggered by a microbial aggression followed by a specific response with local and systemic repercussions. Several authors consider DP as a potential risk factor for development of systemic diseases such as stroke, diabetes, preterm birth, atherosclerosis and cardiovascular diseases. The study of how PD can influence a number of systemic disorders is called a periodontal medicine. The biofilm attached in teeth can release toxic bacterial products biologically active and the periodontum responds to these substances through the inflammation. There is a varied spectrum of periodontal inflammatory responses to microbial attack, from a slight gingivitis to a severe destructive PD. The bacterial products associated with inflammatory mediators can lead the patient to produce a series of systemic responses. The purpose of this review is to discuss the possible biological pathways by which the PD can influence the etiopathogenesis of some diseases and systemic disorders.
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