Abstract
Actin networks polymerize and depolymerize to construct highly organized structures, thereby, endowing the mechanical phenotypes found in a cell. It is generally believed that the amount of filamentous actin and actin network architecture determine cytoplasmic viscosity and elasticity of the whole cell. However, the intrinsic complexity of a cell and other numerous endogenous cellular components make it difficult to study the differential role of distinct actin networks in regulating cell mechanics.
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