Abstract
Plasma FFA level rose in normal weight control and to a lesser degree in obese women when they were exposed to cold at 4–6°C for a period of 30 minutes. This rise, in lean subjects, of plasma FFA was eliminated by pretreatment with either nicotinic acid or propranolol. The response to cold was a biphasic one with an early fall in plasma FFA level followed by a rise. The rise in plasma FFA appears to be catecholamine induced, and the absence of significant change in blood glucose would suggest that norepinephrine may be the stimulus. The biphasic nature of the response to cold suggests a favoring of utilization to mobilization of FFA early and the reverse later. The difference in the response noted in obese women may represent a decreased responsiveness, but possibly may be due to an insulating effect of adipose tissue mass.
Published Version
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a call