Abstract
Two major problems facing diabetic patients are microangiopathy, which leads to renal and visual failure, and accelerated atherosclerosis, which results in ischemia in the extremities, myocardium and other organs, such as kidney and brain. The argument over what is inherited and what is secondary to the glucose-insulin pathophysiology continues, but most authorities agree that the microangiopathy is a result of the abnormal metabolic milieu. This is best shown by the temporal relation between the onset of the juvenile type of diabetes and the various "complications." The earliest demonstrable renal lesions have been only found after several years of diabetes, and . . .
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