Abstract
The composition of refluxed material is an important factor in the pathogenesis of reflux esophagitis. The effect of acid reflux into the esophagus has been well studied and its damage on the esophageal mucosa is clear. There are many studies that suggest the importance of bile salts and pancreatic juice as the cause of esophagitis [1–4], especially when both acid and bile-pancreatic secretion are present. Duodenogastric reflux is currently being widely studied, of interest being the precise role of this factor in the pathogenesis of reflux esophagitis. Increased duodenogastric reflux has been shown in patients with gastroesophageal reflux [3]; however, it is not clear whether duodenogastric reflux is greater in patients with different degrees of damage on the esophageal mucosa.
Published Version
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