Abstract
Glaucoma is one of the leading causes of blindness in the developed world. It is a progressive optic neuropathy where structural loss of retinal ganglion cell (RGC) axons corresponds with functional visual field defect. Glaucoma is distinguished from other optic neuropathies by its selective loss of RGC axons. Superior and inferior peripheral nerve sectors are found to be most vulnerable to pressure induced injury whereas the inner temporal sector is most resilient (Quigley et al., 1988). Pathogenesis behind the preferential axonal damage pattern is still poorly understood.
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