Abstract
During host infection, single-celled apicomplexan parasites like Plasmodium and Toxoplasma use a unique form of locomotion called gliding that differs fundamentally from the swim-or-crawl paradigm of eukaryotic cell motility. Gliding is powered by a thin layer of actin and a specialized myosin sandwiched between the plasma membrane and an inner membranous scaffold. How is this actomyosin network organized to generate coherent traction forces, and drive the diverse cell movements observed during gliding? Here, we used single-molecule imaging to track individual actin filaments and myosin complexes in living Toxoplasma gondii.
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