Abstract
Apoliprotein E4 (apoE4) is the strongest genetic risk factor for Alzheimer’s Disease although the detailed method of action is still unclear. Kim et al (JEM 209, 2149) demonstrated that a monoclonal anti-apoE4 antibody reduced the amyloid deposition in an AD mouse model. These data prompted us to investigate human plasma and the intravenous immunoglobulin G (IVIG) preparation Gammagard Liquid (GGL) for the presence of anti-apoE4 IgG.
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