Abstract
Introduction: Hemorrhagic shock-resuscitation is an activator of the innate immune system with a resulting systemic inflammation response post-injury. the GD T-lymphocyte is a unique CD3+, CD4-/8- lymphocyte that functionally resides between the innate and adaptive arms of the immune system and plays a role in neutrophil activation and end-organ damage in reperfusion. the role of this cell-type in hemorrhagic shock and resuscitation has yet to be fully elucidated. We hypothesized that depletion of GD T-cells would decrease the inflammatory response following hemorrhagic shock-resuscitation.
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