Abstract
Background Delayed enhancement (DE) CMR is the gold standard for detecting irreversibly damaged myocardial tissue (scar). Yet, direct impact of scar on regional systolic and diastolic left ventricular (LV) function is not well understood. Standard tools for LV velocities (Tissue Doppler Imaging) are limited by poor reproducibility and incomplete assessment of all regions and motion directions. Myocardial CMR velocity mapping (MVM) is reproducible, non-invasive, and allows direct measure of myocardial velocities of all LV motion components in all regions. Here, we analyze effects of LV scar burden on myocardial motion.
Highlights
Delayed enhancement (DE) CMR is the gold standard for detecting irreversibly damaged myocardial tissue
DE-CMR: A radiologist assessed whether scar was present in segments from the AHA 16-segment model and classified based on number of segments with scar
Patients with high scar showed reduced regional peak velocities compared to patients with no and low scar
Summary
Delayed enhancement (DE) CMR is the gold standard for detecting irreversibly damaged myocardial tissue (scar). Direct impact of scar on regional systolic and diastolic left ventricular (LV) function is not well understood. Standard tools for LV velocities (Tissue Doppler Imaging) are limited by poor reproducibility and incomplete assessment of all regions and motion directions. Myocardial CMR velocity mapping (MVM) is reproducible, non-invasive, and allows direct measure of myocardial velocities of all LV motion components in all regions. We analyze effects of LV scar burden on myocardial motion
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