Abstract
Rheumatoid arthritis (RA) is driven by antigen-specific Tcell responses targeting the joints. MacDonald etal.1 define the range of dendritic cell (DC) populations within joints of RA patients and highlight specific iDC3 and DC2 populations enriched in inflamed RA synovium that promote Tcell activation as well as tolerogenic AXL+ DC2s in healthy synovium that are lost in RA.
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