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HomeStrokeVol. 45, No. 10Correction Free AccessCorrectionPDF/EPUBAboutView PDFView EPUBSections ToolsAdd to favoritesDownload citationsTrack citationsPermissions ShareShare onFacebookTwitterLinked InMendeleyReddit Jump toFree AccessCorrectionPDF/EPUBCorrection Originally published1 Oct 2014https://doi.org/10.1161/STR.0000000000000047Stroke. 2014;45:e216This article corrects the followingInhibition of Mitochondrial P53 Abolishes the Detrimental Effects of Social Isolation on Ischemic Brain InjuryThe version of the article, “Inhibition of Mitochondrial P53 Abolishes the Detrimental Effects of Social Isolation on Ischemic Brain Injury” by Venna et al that published online ahead-of-print on September 9, 2014, a correction was needed. Figures 1 and 2 were transposed.Download figureDownload PowerPointFigure 1. Social isolation (SI) mice had significantly increased infarct size after stroke in males (A) and in females (C). Values are mean±SEM. At 72 h after stroke, neurological deficit scores were higher in SI mice, suggesting poorer recovery compared with pair-housed (PH) mice in males (B) and in females (D), data presented as box-and-whisker plot. *P<0.05, **P<0.01 indicates statistically significant differences.Download figureDownload PowerPointFigure 2. Western blot images and quantitative analysis showing increased mitochondrial p53 levels 24 h after stroke (COX IV as loading control). The increase is significantly higher in both males (A) and females (C) after stroke and in social isolation (SI) mice compared with stroke pair-housed (PH) mice (n=6/grp). No significant differences were seen in sham groups (n=4/grp). Bcl-2 levels are significantly lower after stroke in SI group compared with PH group (n=6/grp) both in males (B) and females (D). *P<0.05.This correction has been made to the online and print version of the article, which is available at http://stroke.ahajournals.org/content/45/10/3101. Previous Back to top Next FiguresReferencesRelatedDetailsCited By Khanna S, Briggs Z and Rink C (2015) Inducible Glutamate Oxaloacetate Transaminase as a Therapeutic Target Against Ischemic Stroke, Antioxidants & Redox Signaling, 10.1089/ars.2014.6106, 22:2, (175-186), Online publication date: 10-Jan-2015. Related articlesInhibition of Mitochondrial P53 Abolishes the Detrimental Effects of Social Isolation on Ischemic Brain InjuryVenugopal Reddy Venna, et al. Stroke. 2014;45:3101-3104 October 2014Vol 45, Issue 10 Advertisement Article InformationMetrics © 2014 American Heart Association, Inc.https://doi.org/10.1161/STR.0000000000000047 Originally publishedOctober 1, 2014 PDF download Advertisement