Correction to: Platelet Protein Disulfide Isomerase Promotes Glycoprotein Ibα-Mediated Platelet-Neutrophil Interactions Under Thromboinflammatory Conditions.

Abstract

HomeCirculationVol. 139, No. 10Correction to: Platelet Protein Disulfide Isomerase Promotes Glycoprotein Ibα-Mediated Platelet-Neutrophil Interactions Under Thromboinflammatory Conditions Free AccessCorrectionPDF/EPUBAboutView PDFView EPUBSections ToolsAdd to favoritesDownload citationsTrack citationsPermissions ShareShare onFacebookTwitterLinked InMendeleyReddit Jump toFree AccessCorrectionPDF/EPUBCorrection to: Platelet Protein Disulfide Isomerase Promotes Glycoprotein Ibα-Mediated Platelet-Neutrophil Interactions Under Thromboinflammatory Conditions Originally published4 Mar 2019https://doi.org/10.1161/CIR.0000000000000662Circulation. 2019;139:e529This article corrects the followingPlatelet Protein Disulfide Isomerase Promotes Glycoprotein Ibα–Mediated Platelet-Neutrophil Interactions Under Thromboinflammatory ConditionsIn the article by Li et al, “Platelet Protein Disulfide Isomerase Promotes Glycoprotein Ibα-Mediated Platelet-Neutrophil Interactions Under Thromboinflammatory Conditions”, which published online before print on November 14, 2018 (Circulation. 2018. doi: 10.1161/CIRCULATIONAHA.118.036323), corrections are needed.The authors note that a non-final version of the manuscript was incorrectly provided for publication. There were also errors in the representative histograms shown in Figure 3, panels 3H and 3K, which have now been replaced.In the results section, the following sentence appeared: “As analyzed by flow cytometry with different monoclonal anti-GPIbα antibodies, MM2/174 recognizing the elastase-resistant part of GPIbα 36 equally detected WT and mutant GPIbα on CHO cells with two populations (Figure 3H-I).”And has now been updated to:As analyzed by flow cytometry with different monoclonal anti-GPIbα antibodies, MM2/174 recognizing the elastase-resistant part of GPIbα 36 equally detected WT and mutant GPIbα on CHO cells (Figure 3H-I).Additionally, the following sentences were added to the discussion section:“Furthermore, it is noticeable that although Cys209Ser mutation in the GPIbα gene has been associated with Bernard-Soulier syndrome45 and Cys209Ser mutant GPIbα was not transiently expressed in CHO cells already expressing GPIbβ and GPIX,46 under our conditions the mutant protein along with GPIbβ and GPIX was expressed on the surface of CHO cells as detectable by anti-GPIbα antibodies or vWF binding. The reason for these apparent differences between the studies is not known.”These corrections have been made to the current online version of the article, which is available at https://www.ahajournals.org/doi/pdf/10.1161/CIRCULATIONAHA.118.036323.Footnoteshttps://www.ahajournals.org/journal/circ Previous Back to top Next FiguresReferencesRelatedDetailsRelated articlesPlatelet Protein Disulfide Isomerase Promotes Glycoprotein Ibα–Mediated Platelet-Neutrophil Interactions Under Thromboinflammatory ConditionsJing Li, et al. Circulation. 2019;139:1300-1319 March 5, 2019Vol 139, Issue 10 Advertisement Article InformationMetrics © 2019 American Heart Association, Inc.https://doi.org/10.1161/CIR.0000000000000662PMID: 30830803 Originally publishedMarch 4, 2019 PDF download Advertisement