Correction: Corrigendum: Dendritic cells from aged subjects contribute to chronic airway inflammation by activating bronchial epithelial cells under steady state

Abstract

Correction to: Mucosal Immunology advance online publication 23 April 2014; doi:10.1038/mi.2014.28 In this article, published on 23 April 2014, the fifth author's name was misspelled as ‘BenMohamad’. The correct spelling is ‘BenMohamed’. The authors regret the error. Correction to: Mucosal Immunology advance online publication 23 April 2014; doi:10.1038/mi.2014.28 In this article, published on 23 April 2014, the fifth author's name was misspelled as ‘BenMohamad’. The correct spelling is ‘BenMohamed’. The authors regret the error. Correction to:Mucosal Immunology advance online publication 23 April 2014; doi:10.1038/mi.2014.28 In this article, published on 23 April 2014, the fifth author's name was misspelled as ‘BenMohamad’. The correct spelling is ‘BenMohamed’. The authors regret the error. Dendritic cells from aged subjects contribute to chronic airway inflammation by activating bronchial epithelial cells under steady stateMucosal ImmunologyVol. 7Issue 6PreviewThe mechanisms underlying the increased susceptibility of the elderly to respiratory infections are not well understood. The crosstalk between the dendritic cells (DCs) and epithelial cells is essential in maintaining tolerance as well as in generating immunity in the respiratory mucosa. DCs from aged subjects display an enhanced basal level of activation, which can affect the function of epithelial cells. Our results suggest that this is indeed the scenario as exposure of primary bronchial epithelial cells (PBECs) to supernatants from unstimulated DCs of aged subjects resulted in activation of PBECs. Full-Text PDF Open Archive