Abstract
It has been demonstrated that in anesthetized open-chest dogs pretreated with beta adrenoceptor blocking agents the cardiac sympathetic nerve stimulation produces a decrease in coronary blood flow which is abolished by alpha blockade (1–3). In the same condition a phenomenon of “rebound” increase in coronary blood flow was noticed after cessation of the stimulation (1, 2). The purpose of this communication is to elucidate the mechanism involved in this rebound phenomenon.
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