Abstract
This raises alarms about the presence of persistent target organ injury since the acute phase of COVID-19, or whether previous inflammation may have decompensated the mechanisms that some organs perform in the face of chronic disorders. There is evidence that has reported the compromise of the endothelium and myocardium during the acute phase of the disease, with the presence of previous heart disease being considered a risk factor for severe disease and death.
Highlights
Coronary Microvascular Injury in the COVID-19 Patient: The Need for Imaging Studies in Post-COVID-19 Cardiovascular Syndrome
The neurological phenotype is the most debated [1,2], the evidence points to the fact that the main manifestations of post-COVID 19 patients, regardless of disease severity, are dyspnea, fatigue, tachycardia, and difficulties in performing activities of daily living [5]. This raises alarms about the presence of persistent target organ injury since the acute phase of COVID-19, or whether previous inflammation may have decompensated the mechanisms that some organs perform in the face of chronic disorders
There is evidence that has reported the compromise of the endothelium and myocardium during the acute phase of the disease, with the presence of previous heart disease being considered a risk factor for severe disease and death [6]
Summary
Coronary Microvascular Injury in the COVID-19 Patient: The Need for Imaging Studies in Post-COVID-19 Cardiovascular Syndrome. Paula Marcela Cuéllar-Pinzón, MD1, María Paula Montes-Lozano, MD2, Marco Antonio SuárezFuentes, MD3, Miller Jesús Melo-Mora, MD4 and Michael Gregorio Ortega-Sierra, MD5*
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