Abstract
RATIONALE: Atopy in early life may be modulated by the expression and stimulation of TLRs on immunocompetent cells. Infants at high risk for developing atopy demonstrate phenotypic alterations of their CB progenitor cell hemopoietic cytokine receptors (HCR). Since hemopoietic mechanisms are involved in atopic development and maintenance, we investigated the co-expression of TLR and HCR on CB progenitors.
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