Abstract
Broken heart syndrome, stress cardiomyopathy or Takotsubo Syndrome (TTS) first described in 1990 is relatively common in post-menopausal women.1 It is believed to be due to coronary microvascular dysfunction (CMD) and catecholamine surge.1 Although epicardial vasospasm at presentation as well as with acetylcholine provocation are well-documented.1-3 There is hence an emerging alternative view that TTS could have a primary epicardial vasospastic aetiology.2 In which case CMD is a transient epiphenomenon that also convalesces with resolution of left ventricular dysfunction.
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