Abstract

Recovery from exercise-induced fatigue is crucial for subsequent performance. Self-myofascial release (SMR) using a foam roller is an alternative to active recovery (AR). This study aims to compare the effects of passive recovery (PR), AR, and SMR on blood lactate [La-] removal and total quality of recovery (TQR). Twenty-two well trained male athletes (age: 22.6±2.9 years) underwent three testing sessions conducted 72 hours apart but at the same time of each day in a randomized order. After determining resting [La-] and heart rate (HR), the subjects completed a Wingate anaerobic test (WAnT), triggering muscular fatigue. HR and [La-] were measured threeminutes after the WAnT, following which the subjects underwent one of the three different recovery interventions over 15 minutes: PR (lying supine), AR (cycling at 40% of the estimated maximum HR of the respective subject), and SMR (using foam roller on lower extremity muscles). After each recovery intervention, [La-], HR, and TQR were measured. There was no statistically significant difference in [La-] and HR values obtained before the WAnT test (p=0.368, p=0.691, respectively) and right after the WAnT test (0.264, p=0.629) Both AR and SMR were more effective than PR for [La-] removal and obtaining a higher TQR (p<0.001). However, SMR and AR were not superior to one another for blood [La-] removal (p>0.05). In contrast, a significantly higher TQR was observed with SMR than AR and PR (p<0.001). Athletes can apply AR or SMR to recover from strenuous exercise. SMR can be an alternative to PR and AR as a recovery tool.

Highlights

  • Exercise-induced muscle fatigue is defined as the decreased ability to generate appropriate amounts of muscle force or power during on-going contractile activity (Finsterer, 2012)

  • Both active recovery (AR) and Self-myofascial release (SMR) were more effective than passive recovery (PR) for [La-] removal and obtaining a higher total quality of recovery (TQR) (p

  • When the normality condition was not provided, the Friedman Test was used for variance analysis and the Wilcoxon signed-rank test was performed for paired comparisons

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Summary

Introduction

Exercise-induced muscle fatigue is defined as the decreased ability to generate appropriate amounts of muscle force or power during on-going contractile activity (Finsterer, 2012). Exercise-induced alterations in muscle homeostasis, including hydrogen ion (H+) accumulation, potassium loss, depletion of high-energy phosphates (ATP and creatine phosphate) and glycogen, loss of calcium homeostasis, or local ischemia, may be some of the causative factors associated with disruption of the muscle excitation-contraction cycle during intense exercise and in post-exercise muscle fatigue (Mika et al, 2007; Steele et al, 2003). It has been known that high-intensity exercise results in increased levels of both intramuscular and circulating levels of lactate [La-] (Connolly et al, 2003) This increase in [La-], reflecting H+ concentration, has been shown to inhibit contractile performance and cause premature fatigue (Connolly et al, 2003; Corder et al, 2000)

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