Abstract
This article is devoted to the disbalance of glycemic and mineral hemostasis in the etiopathogenetic mechanism of neonatal seizures. The purpose of the article: to assess the state of glycemic and mineral hemostasis in patients with neonatal convulsions. Materials and methods. 73 patients with neonatal convulsions were under observation. A blood glucose, sodium, potassium, magnesium and calcium content were investigated in all patients. Results. Among patients with neonatal convulsions hypoglycemia was detected 34.3±5.6%, hypocalcemia – 34.3±5.6%, hypomagnesaemia – 9.6±3.1%, combination of hypocalcemia with hypomagnesemia – 11.0±3.7%, hyponatremia – 11.0±3.7%, hyperkalemia and hypokalemia – 6.9±3.0%, and hypernatremia – 4.1±2.3%. Conclusion. Depending on the cause of neonatal seizures, the characteristics of glycemic and mineral hemostasis change.
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