Abstract
Recent developments in the field of hemostasis and thrombosis have brought to light the importance that the fibrin clot structure plays in determining the risk for cardiovascular diseases. Levels of circulating blood factors such as fibrinogen, calcium, thrombin, factor XIII, and plasminogen, to name a few, are very sensitive to change and play an important role in the final structure of a blood clot. Aberrant clots made from densely packed, small-diameter fibrin fibers are harder to break down than normal clots with larger fibers and pores, and can lead to serious health risks when left untreated. Clot-altering medications can help to elucidate some of the problems associated with abnormal clot structure. Here we will discuss normal formation of fibrin clots and how the structure of these clots can change in relation to disease and treatment.
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