Abstract
IgA nephropathy (IgAN) is the most common pattern of glomerulonephritis in much of the world and remains an important cause of chronic kidney disease and kidney failure. Much progress has been made in our understanding of the genetic and biochemical pathogenic basis of the disease. The Oxford Classification, a histopathology scoring system developed for IgAN in 2009, is widely validated and adopted. Current treatment of IgAN remains generic to many chronic kidney diseases and glomerulonephritides, while optimal treatment strategies and emerging therapeutic approaches are continually being developed and tested in clinical trials. IgA vasculitis (or Henoch-Schönlein purpura) is a systemic form of vasculitis involving the kidney, gut, skin, and joints but shares many distinct clinical features with IgAN in its kidney involvement.
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