Abstract
Hearing loss is the dominant cause of tinnitus and hyperacusis. Sensorineural hearing loss is accompanied by loudness recruitment that originates from the synchronous firing of many nerve fibers onto nonprimary cells in the ventral cochlear nucleus. Hyperacusis may be related to so-called overrecruitment. Tinnitus manifests itself as phantom percept, where the tinnitus pitch is reflected in the tinnitus spectrum which is often the inverse of the hearing loss frequencies. Tinnitus does occur in so-called clinical normal hearing, i.e., thresholds up to 8kHz below 25dB hearing loss, but often reflect the hearing loss at frequencies above 8kHz.
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