Abstract
During treatment with cephaloridine in 362 patients, 81 had renal impairment with increased blood urea nitrogen levels. In 69 of these, the elevation was secondary to inadequate hydration, obstructive uropathy, or increased sources of nitrogenous products. In 12 patients, acute tubular necrosis was secondary to shock. An additional 21 patients with normal renal function received 6 to 9 gm daily for as long as 52 days without evidence of renal injury. In no instance could cephaloridine be implicated as causing or contributing to renal dysfunction.
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