Abstract
Mechanisms for the myocardial protective effect of TL: included improvement of erythrocyte deformability, inhibition of adrenaline-induced platelet aggregation, endothelium-dependent vasorelaxant effect and suppression of neutrophil adhesion to endothelium. Biochemical studies showed that .TL increased cyclic GMP through a stimulation of nitric oxide synthesis. In addition, TL scavenged free radical formation in neutrophils. These beneficial effects of TL on cardiovascular system was not mediated through linoleic acid which was derived from hydrolysis of TL.
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