Abstract
Carbon monoxide (CO) poisoning is one of the most common types of poisoning in the USA and can be either acute or chronic. CO poisoning is diagnosed through examination of the patients’ history and is confirmed by an elevated carboxyhemoglobin level. The symptoms of CO poisoning are often nonspecific including headache, nausea, vomiting, or other flu-like symptoms which are often misdiagnosed. Many patients with CO poisoning develop new cognitive impairments that can occur immediately after the poisoning or can appear within 40 days of the CO poisoning known as delayed neurologic sequelae. Individuals with CO poisoning develop new morbidities including cognitive impairments and psychological disorders (depression and anxiety). Cognitive impairments occur in a variety of cognitive domains and vary in severity. This chapter will describe the cognitive impairments following CO poisoning, mechanism of injury including the role of hypoxia, and treatment for cognitive impairments.
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