Calcium overload and ischemic myocardial injury.

Abstract

HomeCirculationVol. 83, No. 2Calcium overload and ischemic myocardial injury. Free AccessAbstractPDF/EPUBAboutView PDFSections ToolsAdd to favoritesDownload citationsTrack citationsPermissions ShareShare onFacebookTwitterLinked InMendeleyReddit Jump toFree AccessAbstractPDF/EPUBCalcium overload and ischemic myocardial injury. J D Marsh and T S Smith J D MarshJ D Marsh Search for more papers by this author and T S SmithT S Smith Search for more papers by this author Originally published1 Feb 1991https://doi.org/10.1161/01.CIR.83.2.709Circulation. 1991;83:709–711 Previous Back to top Next FiguresReferencesRelatedDetailsCited By Nemade H, Chaudhari U, Acharya A, Hescheler J, Hengstler J, Papadopoulos S and Sachinidis A (2018) Cell death mechanisms of the anti-cancer drug etoposide on human cardiomyocytes isolated from pluripotent stem cells, Archives of Toxicology, 10.1007/s00204-018-2170-7, 92:4, (1507-1524), Online publication date: 1-Apr-2018. Givvimani S, Pushpakumar S, Metreveli N, Veeranki S, Kundu S and Tyagi S (2015) Role of mitochondrial fission and fusion in cardiomyocyte contractility, International Journal of Cardiology, 10.1016/j.ijcard.2015.03.352, 187, (325-333), Online publication date: 1-May-2015. Wang X, Wang L, Fu C, Zhang P, Wu Y and Ma J (2014) Ranolazine Attenuates the Enhanced Reverse Na+-Ca2+ Exchange Current via Inhibiting Hypoxia-Increased Late Sodium Current in Ventricular Myocytes, Journal of Pharmacological Sciences, 10.1254/jphs.13202FP, 124:3, (365-373), . Tang Q, Ma J, Zhang P, Wan W, Kong L and Wu L (2012) Persistent sodium current and Na+/H+ exchange contributes to the augmentation of the reverse Na+/Ca2+ exchange during hypoxia or acute ischemia in ventricular myocytes, Pflügers Archiv - European Journal of Physiology, 10.1007/s00424-011-1070-y, 463:4, (513-522), Online publication date: 1-Apr-2012. Zheng J, Ma J, Zhang P, Hu L, Fan X and Tang Q (2009) Milrinone inhibits hypoxia or hydrogen dioxide-induced persistent sodium current in ventricular myocytes, European Journal of Pharmacology, 10.1016/j.ejphar.2009.06.021, 616:1-3, (206-212), Online publication date: 1-Aug-2009. Davidson S and Yellon D (2005) The role of nitric oxide in mitochondria . Focus on “Modulation of mitochondrial Ca 2+ by nitric oxide in cultured bovine vascular endothelial cells” , American Journal of Physiology-Cell Physiology, 10.1152/ajpcell.00277.2005, 289:4, (C775-C777), Online publication date: 1-Oct-2005. Caesar R (1999) Herz Pathologie, 10.1007/978-3-642-58620-0_7, (103-276), . Chávez E, Téllez F, Pichardo J, Milán R, Cuéllar A, Carbajal K and Cruz D (1996) On the protection by ketorolac of reperfusion-induced heart damage, Comparative Biochemistry and Physiology Part C: Pharmacology, Toxicology and Endocrinology, 10.1016/S0742-8413(96)00058-8, 115:1, (95-100), Online publication date: 1-Sep-1996. Rose U, Couwenberg P, Jansen J, Bindels R and Van Os C (1994) Effects of substrate-free anoxia and veratridine on intracellular calcium concentration in isolated rat ventricular cardiomyocytes, Pflügers Archiv, 10.1007/BF00374851, 428:2, (142-149), Online publication date: 1-Sep-1994. Nakamura T, Goda K, Okamoto T, Kishi T, Nakamura T and Goshima K (1993) Contractile and morphological impairment of cultured fetal mouse myocytes induced by oxygen radicals and oxidants. Correlation with intracellular Ca2+ concentration., Circulation Research, 73:4, (758-770), Online publication date: 1-Oct-1993. Thornell L, Holmbom B, Eriksson A, Reiz S, Marklund S and N�slund U (1992) Enzyme and immunohistochemical assessment of myocardial damage after ischaemia and reperfusion in a closed-chest pig model, Histochemistry, 10.1007/BF00271069, 98:6, (341-353), . Miyata H, Lakatta E, Stern M and Silverman H (1992) Relation of mitochondrial and cytosolic free calcium to cardiac myocyte recovery after exposure to anoxia., Circulation Research, 71:3, (605-613), Online publication date: 1-Sep-1992. Hayashida K, Miyara S, Shinozaki K, Takegawa R, Yin T, Rolston D, Choudhary R, Guevara S, Molmenti E and Becker L (2021) Inhaled Gases as Therapies for Post–Cardiac Arrest Syndrome: A Narrative Review of Recent Developments, Frontiers in Medicine, 10.3389/fmed.2020.586229, 7 February 1, 1991Vol 83, Issue 2 Advertisement Article InformationMetrics Copyright © 1991 by American Heart Associationhttps://doi.org/10.1161/01.CIR.83.2.709 Originally publishedFebruary 1, 1991 PDF download Advertisement