Abstract
Data on in vivo experiments on mice exposed to Cd with the attempt to correlate serum trypsin inhibitory capacity (TIC) changes with morphologic alterations is presented. The experimental data obtained indicate that cadmium chloride produced a significant reduction in the serum antitrypsin activity in mice. The data further indicates that the dimunition of serum TIC due to acute Cd exposure is a transient phenomenon but appears to produce lung damage rapidly as shown by morphologic examination. (JMT)
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