Brought in by force: AMPK, TBC1D1, and contraction-stimulated glucose transport in skeletal muscle

Abstract

transient intracellular calcium oscillations were the first signaling mediator described to regulate contraction-stimulated glucose transport in skeletal muscle ([6][1]). Thirty years later, the discoveries, that 5′-AMP-activated protein kinase (AMPK) activator 5-aminoimidazole-4-carboxamide 1-β-