Abstract
Abstract Blood samples were withdrawn from dorsal aortic cannulae in shortfin eels before, during, and after acute exposure to hypoxia and to forced swimming. Both hypoxia (PIO2 < 40 mm Hg) and forced swimming at 24 cm s‐1 abolished apnoeic periods and increased ventilation. The fall in PaCO2with hyperventilation, raised pH and thus increased the oxygen carrying capacity of arterial blood. The Root effect influences shortfin eel blood up to pH 8.0, at 17°C and has an effect on arterial oxygen content. It cannot be responsible for all of the increased oxygen content during swimming and in recovery from swimming and hypoxia; ventilatory and/or circulatory changes must account for the rest. Hypoxia, but not swimming, produced a rise in dorsal aortic blood pressure. The bradycardia of hypoxia was deeper than that which accompanies apnoeic periods in air‐saturated water.
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callMore From: New Zealand Journal of Marine and Freshwater Research
Disclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
