Abstract
Etiolated bean hypocotyls underwent necrosis and cellular browning following infection by tobacco necrosis virus. This was accompanied by the production of many phenolic compounds, some of which were shown to be highly antifungal in assays of fungal growth on thin-layer plates. Four compounds, phaseollin, phaseollidin, phaseollinisoflavan and kievitone, were isolated and identified. Their minimum lethal doses towards spore germination of Colletotrichum lindemuthianum were 2, 2, 2 and 20 μg/ml respectively. In addition, following their isolation in high yield from virus-infected tissue, they were also demonstrated in tissue infected with C. lindemuthianum. They have thus been referred to as phytoalexins. The use of virus-infected tissue as a source of new phytoalexins, the rôle of these compounds in disease resistance and their possible importance in explaining virus-induced resistance to fungal pathogens are discussed.
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