Abstract
Objective To evaluate the efficacy of Nd:YAG laser associated with calcium-phosphate desensitizing pastes on dentin permeability and tubule occlusion after erosive/abrasive challenges.Methodology Dentin specimens were exposed to 17% ethylene diamine tetra-acetic acid (EDTA) solution for 5 min and randomly allocated into five groups: G1, control (no treatment); G2, Nd:YAG laser (1 W, 10 Hz, 100 mJ, 85 J/cm2); G3, Laser + TeethmateTM Desensitizer; G4, Laser + Desensibilize Nano P; and G5, Laser+Nupro®. Specimens underwent a 5-day erosion-abrasion cycling. Hydraulic conductance was measured post-EDTA, post-treatment, and post-cycling. Post-treatment and post-cycling permeability (%Lp) was calculated based on post-EDTA measurements, considered 100%. Open dentin tubules (ODT) were calculated at the abovementioned experimental moments using scanning electron microscopy and ImageJ software (n=10). Data were analyzed using two-way repeated measures ANOVA and Tukey’s test (α=0.05).Results G1 presented the highest %Lp post-treatment of all groups (p<0.05), without significantly differences among them. At post-cycling, %Lp significantly decreased in G1, showed no significant differences from post-treatment in G3 and G4, and increased in G2 and G5, without significant differences from G1 (p>0.05). We found no significant differences in ODT among groups (p>0.05) post-EDTA. At post-treatment, treated groups did not differ from each other, but presented lower ODT than G1 (p<0.001). As for post-cycling, we verified no differences among groups (p>0.05), although ODT was significantly lower for all groups when compared to post-EDTA values (p<0.001).Conclusion All treatments effectively reduced dentin permeability and promoted tubule occlusion after application.Combining Nd YAG laser with calcium-phosphate pastes did not improve the laser effect. After erosive-abrasive challenges, treatments presented no differences when compared to the control.
Highlights
Dentin hypersensitivity (DH) is a common painful condition defined as a short and acute pain resulting from exposed dentin in response to external stimuli, such as thermal, osmotic, tactile, chemical, or evaporative, and unrelated to any other dental disease or defect.1 DH prevalence may vary widely among studies, but can be as high as 89.1%.2 Studies have demonstrated that sensitive teeth have more exposed dentin tubules and with larger diameter than those of non-sensitive teeth, so that the patency of dentine tubules is an important and necessary condition for DH to occur.3 Formulated by Brännström and Aström4 in 1972, the hydrodynamic theory postulates that nociceptors located at the pulp-dentin interface can be activated by stimuli-induced changes in dentin fluid flow
Dentin specimens were exposed to 17% ethylene diamine tetra-acetic acid (EDTA) solution for 5 min and randomly allocated into five groups: G1, control; G2, Nd:YAG laser (1 W, 10 Hz, 100 mJ, 85 J/cm2); G3, Laser + TeethmateTM Desensitizer; G4, Laser + Desensibilize Nano P; and G5, Laser+Nupro®
We found no significant differences for dentin permeability among desensitizing treatments (p=0.139), but experimental moments (p
Summary
Dentin hypersensitivity (DH) is a common painful condition defined as a short and acute pain resulting from exposed dentin in response to external stimuli, such as thermal, osmotic, tactile, chemical, or evaporative, and unrelated to any other dental disease or defect. DH prevalence may vary widely among studies, but can be as high as 89.1%.2 Studies have demonstrated that sensitive teeth have more exposed dentin tubules and with larger diameter than those of non-sensitive teeth, so that the patency of dentine tubules is an important and necessary condition for DH to occur. Formulated by Brännström and Aström in 1972, the hydrodynamic theory postulates that nociceptors located at the pulp-dentin interface can be activated by stimuli-induced changes in dentin fluid flow. Formulated by Brännström and Aström in 1972, the hydrodynamic theory postulates that nociceptors located at the pulp-dentin interface can be activated by stimuli-induced changes in dentin fluid flow. Based on this theory, occluding dentinal tubules and reducing dentin permeability are deemed a rational approach to avoid or disable triggers that cause pain. Associating treatments has shown to reach superior results in tubule occlusion, such as the use of high-power lasers along with desensitizing agents.. Associating treatments has shown to reach superior results in tubule occlusion, such as the use of high-power lasers along with desensitizing agents.9 Their effectiveness has not always been shown to be greater in the long-term when compared to a single treatment. Oxalates, strontium, calcium/phosphate formulations, and lasers are some of the agents used to manage DH. For being able to occlude dentinal tubules by mineral deposition, calcium and phosphatebased products are promising options. efficiently used to promote immediate relief, some of these agents lack the necessary ability to resist erosive-abrasive challenges. Associating treatments has shown to reach superior results in tubule occlusion, such as the use of high-power lasers along with desensitizing agents. their effectiveness has not always been shown to be greater in the long-term when compared to a single treatment.
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