Abstract
We examined the methodological approach to the assessment of monosodium glutamate intake. The high carbohydrate and low fat consumption characteristic of this study population would be conducive to the development of metabolic syndrome. However, anomalies in the assessment of dietary information limits conclusion to a causal link of monosodium glutamate to metabolic syndrome and overweight because the study lacks data on the main dietary patterns of consumption. Given the current paucity of data from human studies on monosodium glutamate intake and risk, more studies with robust methodology are required to assess causal links to disease.
Highlights
We examined the methodological approach to the assessment of monosodium glutamate intake
Letters to the editor Dear Editor, The paper by Insawang et al, concerning monosodium glutamate (MSG) intake and its association with metabolic syndrome (Met-S) in a rural Thai population is the centre of a current debate [1,2,3]
They estimated for every 1 g increase in MSG consumption, Met-S risk increased with an odds ratio (OR) of 1.14 (CI 1.12-1.28) or being overweight with an OR of 1.16 (CI 1.04-1.29)
Summary
We examined the methodological approach to the assessment of monosodium glutamate intake. Letters to the editor Dear Editor, The paper by Insawang et al, concerning monosodium glutamate (MSG) intake and its association with metabolic syndrome (Met-S) in a rural Thai population is the centre of a current debate [1,2,3]. They estimated for every 1 g increase in MSG consumption, Met-S risk increased with an odds ratio (OR) of 1.14 (CI 1.12-1.28) or being overweight with an OR of 1.16 (CI 1.04-1.29).
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