Abstract
1. An experimental model of an artificial-heart heat exchanger has been developed which preserves thrombosis-resistant properties for long periods of time. 2. An analytic expression has been derived that relates the maximum blood temperature rise, the quantity of heat power released to the blood, the length of the heated section of the heat exchanger, and the Reynolds number (640<Re<1050). 3. The specificity of the local thermal effect on the fibrinothrombocytic structure in vivo has been revealed. 4. Supplemental thermal loads on a homoisothermal organism amountingto one-half of its normal metabolism has no significant effect on the chemical thermogenesis structure. 5. The supplemental heat is dissipated by the respiratory system.
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