Abstract
Drought and salinity alter redox homeostasis by disrupting basic metabolic processes, such as photosynthesis, respiration, photorespiration, among others, and ultimately causing nonspecific oxidative damage. Information-rich redox buffers in the form of low-molecular-weight antioxidants combined with several other antioxidative enzymes combat those adverse changes. Significantly, antioxidants have been found to provide essential information on the redox state and to influence gene expression under dehydration and salinity stress, which offer protection and stress acclimation. A growing body of evidence strongly suggests the importance of the role of redox homeostasis under salinity and dehydration stress in which the reactive oxygen species (ROS) and antioxidants interact at the metabolic interface for a signal derived from unfavorable environmental cues. This review attempts to detail the role of redox regulation and antioxidant signaling in response to dehydration and salinity stress. In addition, it elaborates our current understanding of ROS–antioxidant interactions in sensing the salinity and dehydration stresses and initiating the signaling necessary for survival.
Published Version
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