Abstract
The diaschisis theory of recovery of function was directly tested for the first time. Using the dentate gyrus in adult rats as the model system, extracellular field potentials elicited by commissural stimulation were monitored in both acute and chronic animal preparations before and from 0.5 hr to 11 days following the lesion. The lesion resulted in the loss of the commissurally elicited long-latency potential but did not disrupt the short-latency monosynaptic potential. No changes in latency, amplitude, form, or stimulus threshold in the monosynaptic potential could be detected.
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