Abstract
Allylisopropylacetamide (AIA) administered to rats, increases the liver δ-aminolevulinic acid (ALA) synthetase activity and porphyrin values elevate plasma triglyceride levels and decreases plasma-free fatty acids. 3,5-Dimethylisooxazole (3,5-DMl) markedly lowers the liver ALA synthetase activity, porphyrin levels and also decreases plasma triglyceride levels in AIA-treated rats. 3,5-DMI exerts its antiporphyric activity probably by increasing the protein catabolism.
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