Abstract
Colorectal cancer (CRC) ranks among the leading causes of cancer death worldwide. The identification of genes that influence colorectal cancer susceptibility and progression therefore remains an essential goal of basic and clinical research. The majority of CRC tumours are microsatellite stable and characteristically show chromosomal instability with losses of large chromosomal regions. Specific patterns of structural chromosomal aberrations and oncogene amplification have been associated with this tumour type.
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