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https://doi.org/10.1136/annrheumdis-2015-eular.3255
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Cite Journal: Annals of the Rheumatic Diseases |  Publication Date: Jun 1, 2015 |
Affiliation: Center for Rheumatology, Ajou University
BackgroundCXCL10 is produced in response to interferon-γ, and tumor necrosis factor-α triggers the accumulation of activated lymphocytes via interaction with a specific receptor, CXCR3, creating an amplification feedback loop. CXCL13...
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