Abstract
A small Acinetobacter plasmid carrying the tet39 tetracycline resistance determinant.
Highlights
5 Long SW, Olsen RJ, Mehta SC et al PBP2a mutations causing high-level ceftaroline resistance in clinical methicillin-resistant Staphylococcus aureus isolates
In Acinetobacter baumannii tetracycline resistance is often caused by tetA(A) and tetA(B), which encode efflux pumps,[1,2] and occasionally by tet(M), which encodes a ribosomal protection protein.[1]
A novel determinant, tet[39], was found in tetracycline-resistant Acinetobacter strains recovered from freshwater fish farms in Denmark[3] and Thailand[4] and from a clinical Acinetobacter calcoaceticus/baumannii complex isolate recovered from human urine in 1986.3 The tetA39 gene encodes an efflux pump that was reported to confer resistance to tetracycline but not minocycline.[3]
Summary
5 Long SW, Olsen RJ, Mehta SC et al PBP2a mutations causing high-level ceftaroline resistance in clinical methicillin-resistant Staphylococcus aureus isolates. A small Acinetobacter plasmid carrying the tet[39] tetracycline resistance determinant In Acinetobacter baumannii tetracycline resistance is often caused by tetA(A) and tetA(B), which encode efflux pumps,[1,2] and occasionally by tet(M), which encodes a ribosomal protection protein.[1] In addition, a novel determinant, tet[39] (tetA39-tetR39), was found in tetracycline-resistant Acinetobacter strains recovered from freshwater fish farms in Denmark[3] and Thailand[4] and from a clinical Acinetobacter calcoaceticus/baumannii complex isolate recovered from human urine in 1986.3 The tetA39 gene encodes an efflux pump that was reported to confer resistance to tetracycline but not minocycline.[3]
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