Abstract
An explanation is sought for the normal stability of the blood pressure and the variations found in different forms of phaeochromocytoma. A rather simple scheme of reactions involving enzymic degradation of pressor amines and another scheme for production and inactivation of the deaminating enzyme is proposed. By applying the principle of the steady state to the combination of these reactions many clinical facts are explained. Quite similar reaction mechanisms may help to explain other physiological and pathophysiological phenomena.
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