Abstract
Background and Aims : Hypercholesterolemia-driven atherosclerosis is a systemic and chronic inflammatory disease propagated in part by monocytes and macrophages. Yet, our knowledge on gene regulation in these cells over time and in different tissues is limited. With spatio-temporal transcriptomic profiling, we aimed to characterize tissue macrophages during atherosclerosis, which will aid in screening for novel atheromatous plaque macrophage- and disease stage-specific therapeutic targets.
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