Abstract
The research article entitled “Electroacupuncture (EA) attenuates ketamine-induced neuronal injury in the locus coeruleus of rats through modulation of the CAMK II/CREB pathway” makes a substantial contribution to our comprehension of the neuroprotective effects associated with EA. In this commentary, our objective is to intricately expand upon the discourse initiated by the focal article, scrutinizing recent advancements in the field and subjecting pivotal issues to critical analysis. By delving into the nuanced implications of the findings and elucidating potential trajectories for future exploration, we intend to cultivate a more profound insight into the therapeutic prowess of EA concerning ketamine-induced neuronal injury.
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