Abstract
Publisher Summary This chapter describes the efforts in evaluating beta-adrenergic influences on renal handling of sodium and water during a behavioral task. The chapter also presents an overview of two experiments. In the first, it has been demonstrated that a shock avoidance task facilitated sodium retention, while the second demonstrated that a beta-adrenergic mechanism was involved in this effect. This research stems from the interest in the delineation of beta-adrenergic influences on the myocardium and vasculature in behavioral paradigms. Both experiments used a dog preparation because control of sodium intake and excretion would be easier to achieve; therefore, any effect of the experimental procedures would be readily observable. The experimental procedures of the two studies are similar with certain exceptions. The possibility that the myocardial and renal effects involve a common neurogenic (beta-adrenergic) influence indicates that the disruption of metabolic processes is pervasive.
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