Abstract
Intrauterine growth restriction (IUGR) is associated with placental insufficiency and infarction. Our previous studies using a rat model of IUGR by bilateral uterine artery ligation (BL) showed that maternal magnesium supplementation significantly reduced IUGR by 64% when assessed 24 hours after. Therefore, we sought to investigate whether oral maternal magnesium (MgCl2) supplementation attenuates placental parenchymal infarctions and apoptosis using the same rat model.
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